Author

Joni M. Mäki

University of OuluORCID 0000-0002-7620-0936Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

Johanna MyllyharjuiD· 2 shared A.S. Rosendahl· 1 shared Antti Railo· 1 shared Anu LaitalaiD· 1 shared Farhan QuadiriD· 1 shared Ilkka MiinalaineniD· 1 shared Jing ZhangiD· 1 shared Karim UllahiD· 1 shared

Activity by Year

2021-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersCancer Biology

Johanna MyllyharjuORCID

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintJul 20, 2026

Inactivation of HIF-P4H-1 Stabilizes IKKα, Modulates Non-Canonical NF-κB Signaling, and Sensitizes Cancer Cells to Cell Death

Karim Ullah, Mizanur Rahman, Farhan Quadir…·Johanna Myllyharju

This study identifies HIF-P4H-1 as a regulator of non-canonical NF-κB signaling by stabilizing IKKα and shows its inhibition sensitizes cancer cells to death.

DOI 10.64898/2026.07.17.739273

humanproteomics

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Molecular BiologyJournal of Investigative DermatologyPeer-reviewedOct 1, 2021

Deletion of hypoxia-inducible factor prolyl 4-hydroxylase 2 in FoxD1-lineage mesenchymal cells leads to congenital truncal alopecia

A.S. Rosendahl, Mia Monnius, Anu Laitala…·Johanna Myllyharju

This study shows that deletion of Hif-p4h-2 in FoxD1-lineage mesenchymal cells causes congenital truncal alopecia by disrupting hair follicle development and signaling pathways.

2 citations

mouseexperimental assay

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