David E. Huber

A Memory Model of Rodent Spatial Navigation: Place Cells are Memories Arranged in a Grid and Grid Cells are Non-spatial

The paper proposes a memory model explaining grid cell responses as a byproduct of hippocampal memory encoding and retrieval, with place cells representing spatial and non-spatial attributes.

A modeling framework for determining modulation of neural-level tuning from non-invasive human fMRI data

This paper introduces a framework to infer neural tuning modulation from fMRI data by comparing theoretical models of how neural responses change across experimental conditions.

Neurocomputational principles underlying the number sense

The paper identifies neurocomputational principles that explain how the brain processes numerosity, showing that center-surround filters, spatial scaling, and divisive normalization account for sensitivity to number and illusions of numerosity.

"Leap before you look": Conditions that promote implicit visuomotor adaptation without explicit learning

The paper investigates conditions under which explicit visuomotor learning can be suppressed, finding that gradual introduction, speed pressure, and visual feedback are necessary for explicit learning to occur.

A Neural Habituation Account of the Negative Compatibility Effect

The paper explains the Negative Compatibility Effect (NCE) using a neural habituation model, showing it results from interactions between rapid perceptual priming and slower response priming.

Neural habituation enhances novelty detection: an EEG study of rapidly presented words

This study investigates how neural habituation affects novelty detection using EEG and machine learning, finding that longer prime durations enhance novelty detection.

Frontal cortex tracks surprise separately for different sensory modalities but engages a common inhibitory control mechanism

The paper investigates how the brain tracks surprise across different sensory modalities and finds separate predictive models for visual and auditory stimuli but a common inhibitory control mechanism.