Author

Pedro Bak‐Gordon

Columbia UniversityORCID 0000-0002-8108-6285Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Molecular Biology· 2

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Organism:human· 2 Approach:statistical model· 2 Research Focus:cell-cell communication· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

James L. ManleyiD· 2 shared Zhaoqi LiuiD· 2 shared Jian ZhangiD· 1 shared Jindou XieiD· 1 shared Minu SeongiD· 1 shared Peiqi XingiD· 1 shared Peter CanolliD· 1 shared

Activity by Year

2024-2025 · peak 1/yr

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7 top collaborators · 15 mutual co-author links

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2 shared papersMolecular Biology

James L. ManleyORCID

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2 papers

Recent·Most cited

Molecular BiologybioRxivPreprintFeb 17, 2025

SUGP1 loss is the sole driver of SF3B1 hotspot mutant missplicing in cancer

Peiqi Xing, Pedro Bak‐Gordon, Jindou Xie…·James L. Manley

The paper identifies SUGP1 loss as the sole driver of SF3B1 hotspot mutant missplicing in cancer, while AQR loss has an indirect effect.

DOI 10.1101/2025.02.17.638713

humanbulk RNA-seq

𝕏 PostBluesky

Molecular BiologyGliaPeer-reviewedOct 24, 2024

Splicing dysregulation in glioblastoma alters the function of cell migration-related genes

Minu Seong, Pedro Bak‐Gordon, Zhaoqi Liu…·James L. Manley

The paper introduces a novel AS analysis method, Semi-Q, to identify GBM-specific splicing events and shows that dysregulated splicing of cell migration-related genes contributes to tumor growth and invasiveness.

7 citations

humanbulk RNA-seq

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