Author

Xin Li

University of CopenhagenORCID 0000-0002-8249-0853Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Xin Li21 papers · Westlake University

Biology Disciplines

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Cancer Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cancer & tumor microenvironment· 1 Approach:genetic perturbation / CRISPR· 1 Organism:human· 1 Assay:whole-genome sequencing· 1

Frequent Co-Authors

André Vidas OlseniD· 1 shared Balca R. MardiniD· 1 shared Claus Storgaard SørenseniD· 1 shared Francesco FaveroiD· 1 shared Francisco German Rodriguez GonzaleziD· 1 shared Joachim WeischenfeldtiD· 1 shared Karolin VoßgröneiD· 1 shared Krushanka Kashyap· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

8 top collaborators · 28 mutual co-author links

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1 shared paperCancer Biology

André Vidas OlsenORCID

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1 paper

Recent·Most cited

Enzyme family-centred approach identifies helicases as recurrent hemizygous tumour suppressor genes

Karolin Voßgröne, Francesco Favero, Krushanka Kashyap, Francisco German Rodriguez Gonzalez, André Vidas Olsen, Xin Li, Balca R. Mardin, Joachim Weischenfeldt, Claus Storgaard Sørensen

The paper identifies helicases as frequently mutated enzyme families in cancer, showing their role in genomic instability and DNA repair defects, with AQR as a novel hemizygous tumor suppressor.

Science Advances · Feb 20, 2026Peer-reviewedCancer Biology