Hannah Brunsdon

The paper shows that loss of MITF activity in zebrafish melanocyte stem cells leads to premature expansion and novel transcriptional states during embryogenesis, with stage-specific effects on regeneration.

The paper develops zebrafish models of PROS to show that PIK3CA mutations cause non-cell-autonomous overgrowth through pan-lineage gene expression and communication changes.

The paper identifies Tfap2b as a marker for melanocyte stem cells in zebrafish, showing their multi-fate potential and role in melanocyte regeneration.

The paper identifies a wound-specific notochord sheath cell subpopulation expressing Wilms Tumor 1b that forms a stopper-like structure during vertebra repair in zebrafish.