Author

James N. Weiss

University of California, Los AngelesORCID 0000-0002-8622-6427Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Biophysics· 1 Systems Biology· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:electrophysiology· 1

Frequent Co-Authors

Alain KarmaiD· 2 shared Marc SantoliniiD· 2 shared Aldons J. Lusis· 1 shared Aldons Jake LusisiD· 1 shared Christoph D. RauiD· 1 shared Clara Yukhtman· 1 shared Colin Rees· 1 shared Jeffrey J. SaucermaniD· 1 shared

Activity by Year

2018-2018 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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2 shared papersBiophysics

Alain KarmaORCID

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2 papers

Recent·Most cited

Variability and compensation of cardiomycoyte ionic conductances at the population level

Colin Rees, Jun-Hai Yang, Marc Santolini, Aldons J. Lusis, James N. Weiss, Alain Karma

The paper quantifies variability in cardiac ion conductances across a genetically diverse mouse population and shows compensatory mechanisms between Ca2+ and K+ currents.

bioRxiv · Mar 15, 2018 · PreprintBiophysics

A personalized, multi-omics approach identifies genes involved in cardiac hypertrophy and heart failure

Marc Santolini, Milagros C. Romay, Clara Yukhtman, Christoph D. Rau, Shuxun Vincent Ren, Jeffrey J. Saucerman, Jessica Wang, James N. Weiss, Yibin Wang, Aldons Jake Lusis, Alain Karma

The paper identifies disease-associated genes missed by traditional methods using a personalized differential gene expression analysis in a genetically diverse mouse model of cardiac hypertrophy and heart failure.

npj Systems Biology and Applications · Feb 20, 2018Peer-reviewedSystems Biology