Author

Andrea J. Wellington

University of ArizonaORCID 0000-0002-8679-9225Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 3

Signature Assays, Organisms & Methods

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Assay:electronic health records· 3 Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1

Frequent Co-Authors

Erika D. EggersiD· 3 shared Diego S. Del Villar· 1 shared Jared T. Hill· 1 shared Michael D. FloodiD· 1 shared Timothy David Maley· 1 shared

Activity by Year

2020-2026 · peak 1/yr

Co-Author Collaboration Network

5 top collaborators · 5 mutual co-author links

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3 shared papersNeuroscience

Erika D. EggersORCID

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3 papers

Recent·Most cited

Optogenetic Isolation of the Amacrine Cell-OFF Bipolar Cell Synapse Shows Selective D1R Control of Glycinergic Transmission

Timothy David Maley, Andrea J. Wellington, Erika D. Eggers

The paper uses optogenetics to isolate the amacrine cell-OFF bipolar cell synapse and shows that D1R selectively reduces glycinergic inhibition.

bioRxiv · Jun 22, 2026 · PreprintNeuroscience

Early Diabetic Ca2+ Handling Impairments in the Rod Bipolar Pathway

Jared T. Hill, Andrea J. Wellington, Diego S. Del Villar, Erika D. Eggers

This study investigates Ca2+ handling impairments in amacrine cells of diabetic mice using optogenetics and pharmacological blockers, finding altered kinetics in diabetic responses to L-type Ca2+ channel inhibition.

bioRxiv · Dec 3, 2025 · PreprintNeuroscience

Impaired light adaptation of ON-sustained ganglion cells in early diabetes is attributable to diminished dopamine D4 receptor sensitivity

Michael D. Flood, Andrea J. Wellington, Erika D. Eggers

This study shows that impaired light adaptation in diabetic mice is due to reduced dopamine D4 receptor sensitivity, not decreased receptor expression.

bioRxiv · Oct 31, 2020 · PreprintNeuroscience