Author

Julia Spanier

Center for Experimental and Clinical Infection ResearchORCID 0000-0002-8797-3563Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Immunology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

Ulrich KalinkeiD· 2 shared Anne KrugiD· 1 shared Antonio Santos‐PeraliD· 1 shared Apurva DhavaleiD· 1 shared Bernadett PályiiD· 1 shared Branka HorvatiD· 1 shared Dániel DériiD· 1 shared Elena WinheimiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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Focal author (center)Selected co-authorORCID co-authorUnlinked name

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2 shared papersMicrobiology

Ulrich KalinkeORCID

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2 papers

Recent·Most cited

Nipah virus Malaysia and Bangladesh strain-induced pathogenesis in mice lacking type I interferon receptor signaling

Lucia Amurri, Olivier Reynard, Ilona Ronco, Dániel Déri, Bernadett Pályi, Julia Spanier, Jennifer Skerra, Olivia Terceve, Thibaut Larcher, Ulrich Kalinke, Zoltán Kis, Branka Horvat, Mathieu Iampietro

This study compares the pathogenesis of NiV-Malaysia and NiV-Bangladesh strains in IFNAR KO mice, showing differential outcomes based on infection route and strain.

PLoS neglected tropical diseases · Aug 20, 2026Peer-reviewedMicrobiology

RIG-I-like receptor-dependent type I Interferon regulates antigen dose and activation in yellow fever vaccine 17D-infected antigen presenting cells

Magdalena Zaucha, Elena Winheim, Antonio Santos‐Peral, Apurva Dhavale, Paul Schwarzlmueller, Frank Dahlström, Giulia Spielmann, Magdalena K. Scheck, Linus Rinke, Yiqi Huang, Varvara Arzhakova, Katharina Eisenächer, Hadi Karimzadeh, Michael Pritsch, Julia Spanier, , Anne Krug

This study shows that type I interferon, induced by RIG-I-like receptors, restricts YF17D replication in antigen-presenting cells, thereby limiting antigen presentation to T cells.

bioRxiv · Oct 8, 2025 · PreprintImmunology