Author

Edward B. Prideaux

University of California San DiegoORCID 0000-0002-8811-869XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Immunology· 1 Systems Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Organism:human· 2 Approach:statistical model· 2 Research Focus:cell-cell communication· 1 Assay:DNA methylation· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Assay:scATAC-seq· 1

Frequent Co-Authors

David L. BoyleiD· 2 shared Gary S. FiresteiniD· 2 shared Jane Hoyt BuckneriD· 2 shared Kevin D. DeaneiD· 2 shared Vernon Michael HolersiD· 2 shared Wei Wang· 2 shared Amy Westermann· 1 shared Andrea E. OchoaiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersImmunology

David L. BoyleORCID

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2 papers

Recent·Most cited

ImmunologyArthritis & RheumatologyPeer-reviewedApr 23, 2026

Epigenetic trajectory predicts development of clinical rheumatoid arthritis in ACPA+ individuals: Targeting Immune Responses for Prevention of Rheumatoid Arthritis (TIP-RA)

Edward B. Prideaux, David L. Boyle, Eunice Choi…·Wei Wang

The paper identifies epigenetic trajectories in ACPA+ individuals that predict RA development, showing dynamic methylation changes in Pre-RA compared to stable methylomes in non-converters and controls.

9 citationstop 1%

humanDNA methylation

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Systems BiologybioRxivPreprintFeb 8, 2025

Multi-lineage transcriptional and cell communication signatures define evolving personalized mechanisms that initiate and perpetuate rheumatoid arthritis

Cong Liu, Z. Audrey Wang, Edward B. Prideaux…·Wei Wang

The paper identifies shared transcriptional and cell communication signatures in RA patients and at-risk individuals, showing how multiple pathogenic mechanisms contribute to disease initiation and progression.

4 citations

humanscATAC-seq

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