Author

Masakazu Yamamoto

University of ConnecticutORCID 0000-0002-8855-4934Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:mouse· 2 Assay:ChIP-seq· 1 Assay:DNA methylation· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Approach:statistical model· 1

Frequent Co-Authors

David J. GoldhameriD· 2 shared Cory J Jubinville· 1 shared Erica Korolev· 1 shared Heather K Jamieson· 1 shared James R. Camp· 1 shared Jelena Erceg· 1 shared Jennifer Cj Chen· 1 shared Katherine Fleck· 1 shared

Activity by Year

2020-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

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2 shared papersDevelopmental Biology

David J. GoldhamerORCID

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2 papers

Recent·Most cited

Developmental BiologybioRxivPreprintJul 30, 2026

Developmental expression of the skeletal muscle determination gene, MyoD, is regulated by novel enhancer elements that interact with the core enhancer and distal regulatory region

Heather K Jamieson, James R. Camp, Katherine Fleck…·David J. Goldhamer

The paper identifies novel enhancer elements that regulate MyoD expression during skeletal muscle development and show they are part of a cis-regulatory network.

DOI 10.64898/2026.07.29.741533

mouseChIP-seq

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Developmental BiologyJournal of Bone and Mineral ResearchPeer-reviewedDec 1, 2020

Over-expression of wild-type ACVR1 in fibrodysplasia ossificans progressiva mice rescues perinatal lethality and inhibits heterotopic ossification

Masakazu Yamamoto, Sean J. Stoessel, Shoko Yamamoto, David J. Goldhamer

This study shows that over-expressing wild-type ACVR1 in FOP mice reduces heterotopic ossification and rescues perinatal lethality.

14 citations

mouseexperimental assay

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