Author

Nadav I. Weinstock

University at Buffalo, State University of New YorkORCID 0000-0002-9065-4176Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Neuroscience· 2

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Lawrence G. WrabetziD· 2 shared M. Laura FeltriiD· 2 shared Alexander Martin RossoriD· 1 shared Conlan KreheriD· 1 shared Daesung ShiniD· 1 shared Dalian DingiD· 1 shared Duc At NguyeniD· 1 shared Edward L. HurleyiD· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersNeuroscience

Lawrence G. WrabetzORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

MPZ-T124M mouse model replicates human axonopathy and suggest alteration in axo-glia communication

Ghjuvan’Ghjacumu Shackleford, Leandro N. Marziali, Yo Sasaki, Nadav I. Weinstock, Alexander Martin Rossor, Nicholas J. Silvestri, Emma R. Wilson, Edward L. Hurley, Grahame J. Kidd, Senthilvelan Manohar, Dalian Ding, Richard Salvi, M. Laura Feltri, Maurizio D’Antonio, Lawrence G. Wrabetz

The paper develops a P0T124M mouse model of CMT2J neuropathy, showing axonal damage without significant myelin defects and altered axo-glia communication.

bioRxiv · May 9, 2022 · PreprintNeuroscience

Brainstem development requires galactosylceramidase and is critical for the pathogenesis of Krabbe Disease

Nadav I. Weinstock, Conlan Kreher, Jacob Favret, Duc At Nguyen, Ernesto Roque Bongarzone, Lawrence G. Wrabetz, M. Laura Feltri, Daesung Shin

This study shows that GALC is critical for brainstem development and KD pathogenesis, with a specific vulnerability period between postnatal days 4-6.

Nature Communications · Oct 23, 2020Peer-reviewedNeuroscience