Author

Emelie Styring

Lund UniversityORCID 0000-0002-9409-2481Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:human· 2 Approach:statistical model· 2 Research Focus:clustering· 1 Research Focus:differential expression· 1 Research Focus:variant calling· 1 Research Focus:variant effect prediction· 1 Assay:whole-exome sequencing· 1 Assay:whole-genome sequencing· 1

Frequent Co-Authors

Daniel BaumhoeriD· 2 shared Karim H. SabaiD· 2 shared Karolin H. NordiD· 2 shared Michaela Nathrath· 2 shared Valeria DifilippoiD· 2 shared Diana C. J. Spierings· 1 shared Felix HaglundiD· 1 shared Felix Haglund de Flon· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 41 mutual co-author links

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2 shared papersGenomics

Daniel BaumhoerORCID

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Genetic profiling of soft tissue and bone tumors using SarcDBase

Valeria Difilippo, Karim H. Saba, Karin Wallander, Emelie Styring, Michaela Nathrath, Daniel Baumhoer, Felix Haglund, Karolin H. Nord

SarcDBase is a tool that extracts and interprets genetic alterations from tumor sequencing data, showing high agreement with curated diagnoses in most cases.

bioRxiv · May 15, 2026 · PreprintGenomics

CDK4 is co-amplified with either TP53 promoter gene fusions or MDM2 through distinct mechanisms in osteosarcoma

Karim H Saba, Valeria Difilippo, Emelie Styring, Jenny Nilsson, Linda Magnusson, Hilda van den Bos, Diana C. J. Spierings, Floris Foijer, Michaela Nathrath, Felix Haglund de Flon, Daniel Baumhoer, Karolin H Nord

This study identifies four subgroups of osteosarcomas based on genomic and transcriptomic analysis, revealing distinct mechanisms of CDK4 amplification in relation to MDM2 or TP53 alterations.

bioRxiv · Mar 13, 2024 · PreprintCancer Biology