Author

Karolin H. Nord

Lund UniversityORCID 0000-0002-2397-2254Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:human· 2 Approach:statistical model· 2 Research Focus:clustering· 1 Research Focus:differential expression· 1 Research Focus:variant calling· 1 Research Focus:variant effect prediction· 1 Assay:whole-exome sequencing· 1 Assay:whole-genome sequencing· 1

Frequent Co-Authors

Daniel BaumhoeriD· 2 shared Emelie StyringiD· 2 shared Felix HaglundiD· 2 shared Karim H. SabaiD· 2 shared Valeria DifilippoiD· 2 shared Diana C. J. SpieringsiD· 1 shared Floris FoijeriD· 1 shared Hilda van den BosiD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 41 mutual co-author links

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2 shared papersGenomics

Daniel BaumhoerORCID

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2 papers

Recent·Most cited

Genetic profiling of soft tissue and bone tumors using SarcDBase

Valeria Difilippo, Karim H. Saba, Karin Wallander, Emelie Styring, Michaela Nathrath, Daniel Baumhoer, Felix Haglund, Karolin H. Nord

SarcDBase is a tool that extracts and interprets genetic alterations from tumor sequencing data, showing high agreement with curated diagnoses in most cases.

bioRxiv · May 15, 2026 · PreprintGenomics

CDK4 is co-amplified with either TP53 promoter gene fusions or MDM2 through distinct mechanisms in osteosarcoma

Karim H. Saba, Valeria Difilippo, Emelie Styring, Jenny Nilsson, Linda Magnusson, Hilda van den Bos, René Wardenaar, Diana C. J. Spierings, Floris Foijer, Michaela Nathrath, Felix Haglund, Daniel Baumhoer, Karolin H. Nord

This study identifies four subgroups of osteosarcomas based on genomic and transcriptomic analysis, revealing distinct mechanisms of CDK4 amplification in relation to MDM2 or TP53 alterations.

npj Genomic Medicine · Sep 25, 2024Peer-reviewedCancer Biology