Author

Stephanie S. Buss

Beth Israel Deaconess Medical CenterORCID 0000-0002-9912-063XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Neuroscience· 2

Signature Assays, Organisms & Methods

Click to filter · or open tag

Assay:electronic health records· 2 Organism:human· 2 Research Focus:benchmarking· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Approach:statistical model· 1

Frequent Co-Authors

Mouhsin M. ShafiiD· 2 shared Peter J. FriediD· 2 shared Aimee Arely Flores‐Sandoval· 1 shared Álvaro Pascual‐LeoneiD· 1 shared Brice PasseraiD· 1 shared Christopher BenwelliD· 1 shared Daniel Zvi PressiD· 1 shared George StothartiD· 1 shared

Activity by Year

2023-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersNeuroscience

Mouhsin M. ShafiORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Neurophysiological signatures of default mode network dysfunction and cognitive decline in Alzheimer disease.

Recep A. Ozdemir, Brice Passera, Peter J. Fried, Daniel Zvi Press, Lynn W. Shaughnessy, Stephanie S. Buss, Mouhsin M. Shafi

This study uses TMS-EEG to identify network-specific hyper-excitability and disrupted connectivity in the default mode network of early Alzheimer's disease, linking these changes to cognitive impairments.

bioRxiv · Sep 16, 2024 · PreprintNeuroscience

Resting-state EEG signatures of Alzheimer's disease are driven by periodic but not aperiodic changes

Martina Kopčanová, Luke Tait, Thomas Donoghue, George Stothart, Laura Jennie Smith, Aimee Arely Flores‐Sandoval, Paula Davila-Pérez, Stephanie S. Buss, Mouhsin M. Shafi, Álvaro Pascual‐Leone, Peter J. Fried, Christopher Benwell

The paper investigates whether Alzheimer's disease EEG changes are due to periodic or aperiodic signal alterations, finding they are purely periodic.

Neurobiology of Disease · Dec 17, 2023Peer-reviewedNeuroscience