Ssang‐Taek Lim

This study shows that collagen-dependent DDR1 ectodomain shedding reduces proliferative signaling in radiation-resistant glioblastoma by decreasing DDR1 phosphorylation and increasing ADAMTS1 activity.

This study identifies SON as a suppressor of circRNA biogenesis in glioblastoma, linking circRNA depletion to PKR signaling and radiation resistance.

This study identifies FAK activity as a mediator of disturbed flow-induced atherosclerosis through the VEGFR2-Cbl-NF-κB signaling pathway in endothelial cells.

This study shows FAK inhibition reduces DNA methylation and reactivates the tumor suppressor DAB2 in breast cancer cells, leading to decreased proliferation.

This study develops a mouse model of ZTTK syndrome by creating Son haploinsufficiency mice, revealing critical roles of SON in organ development and hematopoiesis.