Kyuho Jeong

This study identifies FAK activity as a mediator of disturbed flow-induced atherosclerosis through the VEGFR2-Cbl-NF-κB signaling pathway in endothelial cells.

This study shows FAK inhibition reduces DNA methylation and reactivates the tumor suppressor DAB2 in breast cancer cells, leading to decreased proliferation.

This study develops a mouse model of ZTTK syndrome by creating Son haploinsufficiency mice, revealing critical roles of SON in organ development and hematopoiesis.