Rafael Gonzalez-Ricon

This study shows that maternal IL-17A administration to germ-free mice does not disrupt fetal cortical lamination, suggesting IL-17A alone is insufficient to cause brain abnormalities without microbial signals.

This study shows that influenza A virus infection during pregnancy increases the transfer of maternal bloodborne molecules to fetal tissues, potentially activating proinflammatory pathways in fetal brain regions.

This study investigates how influenza A virus infection during pregnancy affects maternal intestinal immunity and fetal brain development, finding dose-dependent effects without increased IL-17 signaling.