Jung-Mi Lee

This study shows that MLL4 is essential for palatal gland development, as its deletion leads to hypoplasia and ectopic cartilage formation.

This study investigates the role of KMT2D in amelogenesis using a conditional knockout mouse model, revealing its critical role in enamel development and identifying direct target genes involved in ameloblast differentiation.

This study shows p63 uses a skin keratinization mechanism for enamel organ development, revealing a shared pathway in tissue specialization.

This study shows that MLL4 is critical for midpalatal suture development and palate growth, with loss of MLL4 causing hypoplasia, widened suture, and disrupted cartilage development.