Author

Celine Sze Ling Chui

Hong Kong Science and Technology Parks CorporationORCID 0000-0003-1513-8726Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Genetic & Genomic Medicine· 1 Pathology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Assay:electronic health records· 2 Organism:human· 2 Approach:statistical model· 2 Research Focus:cell type annotation· 1 Research Focus:GWAS / association mapping· 1

Frequent Co-Authors

Bin CuiiD· 1 shared Chunyan HuiD· 1 shared Eric Yuk Fai WaniD· 1 shared Esther W. ChaniD· 1 shared George Davey SmithiD· 1 shared Hao Luo· 1 shared Haoyu LiuiD· 1 shared Hong LiniD· 1 shared

Activity by Year

2023-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperGenetic & Genomic Medicine

Bin CuiORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

PathologyChaos An Interdisciplinary Journal of Nonlinear SciencePeer-reviewedFeb 1, 2025

Deciphering the Molecular Mechanism of Post-Acute Sequelae of COVID-19 through Comorbidity Network Analysis

Lue Tian, Eric Yuk Fai Wan, Celine Sze Ling Chui…·Qingpeng Zhang

This study analyzes longitudinal electronic health records to identify comorbidity patterns influenced by COVID-19 and their biological mechanisms through protein-protein interaction networks.

DOI 10.1063/5.0250923

humanelectronic health records

𝕏 PostBluesky

Genetic & Genomic MedicinemedRxivPreprintOct 10, 2023

Mendelian randomization revealing the protective effect of sodium-glucose cotransporter 2 inhibition on prostate cancer with verified evidence from electronic healthcare and biological data

Jie Zheng, Jieli Lu, Jiying Qi…·Yufang Bi

This paper uses Mendelian randomization and electronic health records to show SGLT2 inhibitors reduce prostate cancer risk, with additional evidence from differential expression analysis.

DOI 10.1101/2023.10.10.23296790

humanelectronic health records

𝕏 PostBluesky