Venkataragavan Chandrasekaran

This study shows CD5L deficiency worsens joint damage in RA by promoting inflammation and non-classical monocyte expansion.

The paper introduces a compositional model to predict protein interactions, showing that functional group composition can reliably predict survivin binding without relying on amino acid sequences.

This study shows insulin influences epigenetic regulation of memory T cells in RA via H3K27ac-survivin mechanism, affecting metabolic activity and immune responses.

This study shows survivin interacts with BAF/SWI complex at bivalent chromatin regions to repress DNA repair in IFNγ-stimulated CD4+ T cells, promoting autoimmune profiles.

The paper shows survivin interacts with PRC2, preventing H3K27 methylation and gene silencing in CD4+T cells.