Author

Siyuan Zhao

Rutgers, The State University of New JerseyORCID 0000-0003-2005-9440Unclaimed ORCID profile · follow to get notified of new preprints

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Neuroscience· 1

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Approach:experimental assay· 1 Organism:human· 1

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Tibor RohácsiD· 1 shared Yevgen K. YudiniD· 1 shared

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2020-2020 · peak 1/yr

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1 shared paperNeuroscience

Tibor RohácsORCID

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1 paper

Recent·Most cited

NeuroscienceeLifePeer-reviewedApr 28, 2020

Disease-associated mutations in TRPM3 render the channel overactive via two distinct mechanisms

Siyuan Zhao, Yevgen K. Yudin, Tibor Rohács

The paper shows two disease-associated TRPM3 mutations cause overactivity through distinct mechanisms, with potential therapeutic implications using primidone.

53 citations

humanexperimental assay

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