Maria G. Tektonidou

This study identifies an m6A epitranscriptomic signature in systemic sclerosis that drives profibrotic changes, which can be reversed by METTL3 inhibition.

This study identifies disease-associated alterations in gene co-regulation patterns in SLE, showing fragmentation and redistribution of co-expression domains correlate with disease activity and biological pathways.

This study identifies distinct susceptibility and activity/severity signatures in SLE using transcriptomics and genetics, enabling improved diagnosis and personalized therapy.