Author

András Dávid Tóth

Semmelweis UniversityORCID 0000-0003-2746-9370Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Approach:deep learning· 1 Approach:experimental assay· 1 Approach:statistical model· 1

Frequent Co-Authors

András BallaiD· 2 shared Asuka InoueiD· 2 shared Eszter Soltész‐Katona· 2 shared Gábor TuruiD· 2 shared László HunyadyiD· 2 shared Péter VárnaiiD· 2 shared Susanne ProkopiD· 2 shared Ádám Misák· 1 shared

Activity by Year

2024-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 43 mutual co-author links

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2 shared papersCell Biology

András BallaORCID

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2 papers

Recent·Most cited

Cell BiologyScience SignalingPeer-reviewedJun 25, 2024

Receptor endocytosis orchestrates the spatiotemporal bias of β-arrestin signaling

András Dávid Tóth, Bence Szalai, Orsolya Tünde Kovács…·László Hunyady

The study shows that β-arrestin signaling bias is spatiotemporally determined by ligand dissociation rate and G protein activity at endosomes, not receptor conformation.

18 citations

experimental assaystatistical model

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BiochemistryCell ReportsPeer-reviewedMay 1, 2024

ArreSTick Motif is Responsible for GPCR-beta-Arrestin Binding Stability and Extends Phosphorylation-Dependent beta-arrestin Interactions to Non-Receptor Proteins

András Dávid Tóth, Eszter Soltész‐Katona, Katalin Kis…·Gábor Turu

The paper identifies the arreSTick motif as a key determinant of GPCR-{beta}-arrestin binding stability and shows its presence in non-receptor proteins.

4 citations

deep learningcell type annotation

𝕏 PostBluesky