Author

Rosemary L. Balleine

The University of SydneyORCID 0000-0003-2864-4345Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1 Genetic & Genomic Medicine· 1 Genetics· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Assay:proteomics· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Research Focus:GWAS / association mapping· 1

Frequent Co-Authors

Adel T. ArefiD· 2 shared Peter G. HainsiD· 2 shared Phillip James RobinsoniD· 2 shared Qing ZhongiD· 2 shared Roger R. ReddeliD· 2 shared Zainab NooriD· 2 shared ABCTB Investigators· 1 shared Adinda BateniD· 1 shared

Activity by Year

2022-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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2 shared papersGenetic & Genomic Medicine

Adel T. ArefORCID

Mutual co-authors in cluster

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3 papers

Recent·Most cited

Genetic & Genomic MedicinemedRxivPreprintJul 8, 2025

A proteogenomic approach to identifying a gene signature associated with HOXB13 G84E carrier status in prostate cancer tumours

Kelsie Raspin, Zainab Noor, Adel T. Aref…·Joanne L. Dickinson

The paper identifies a gene and protein expression signature associated with HOXB13 G84E carrier status in prostate cancer using proteogenomic analysis.

DOI 10.1101/2025.07.08.25331050

humanproteomics

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Cancer BiologyCancer DiscoveryPeer-reviewedJun 9, 2025

Federated deep learning enables cancer subtyping by proteomics

Zhaoxiang Cai, Emma L. Boys, Zainab Noor…·Roger R. Reddel

ProCanFDL uses federated deep learning to subtype cancers using proteomic data while preserving privacy, achieving performance gains over local models.

18 citationstop 3%

humanproteomics

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GeneticsBreast Cancer ResearchPeer-reviewedJan 4, 2022

Common variants in breast cancer risk loci predispose to distinct tumor subtypes

Thomas U. Ahearn, Haoyu Zhang, Kyriaki Michailidou…·Peter T. Simpson

The paper identifies breast cancer risk variants associated with tumor subtypes and shows their differential effects on luminal and non-luminal subtypes.

33 citations

humanGWAS summary statistics

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