Author

Shawn B. Bratton

The University of Texas MD Anderson Cancer CenterORCID 0000-0003-2895-5020Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Cancer Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cancer & tumor microenvironment· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

Daric J. WibleiD· 1 shared Dean G. TangiD· 1 shared Manu M. SebastianiD· 1 shared Wen Jess LiiD· 1 shared Xiaozhuo LiuiD· 1 shared

Activity by Year

2025-2025 · peak 1/yr

Co-Author Collaboration Network

5 top collaborators · 10 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCancer Biology

Daric J. WibleORCID

Mutual co-authors in cluster

Author profile →

1 paper

Recent·Most cited

Cancer BiologybioRxivPreprintMay 2, 2025

Loss of ATG5 expression in a subset of human prostate cancers promotes tumor growth through accumulation of p62

Daric J. Wible, Wen Jess Li, Xiaozhuo Liu…·Shawn B. Bratton

The paper shows that loss of ATG5 in prostate cancers leads to tumor growth by accumulating p62, suggesting ATG5 acts as a tumor suppressor in this context.

DOI 10.1101/2025.05.02.651776

humanproteomics

𝕏 PostBluesky