Rhima M. Coleman

This study shows that mismatch in cartilage thickness and mechanical properties at the patellar osteochondral allograft interface leads to localized high-strain regions that may cause graft failure.

This study develops modular, vascularized hypertrophic cartilage constructs for bone tissue engineering that support simultaneous mineralization and vessel formation without exogenous supplements.

The paper designs a synthetic gene circuit to autonomously regulate RUNX2 activity during chondrogenesis, enhancing matrix synthesis and resisting ECM degradation.