Author

Massimo Onor

Institute for the Chemistry of OrganoMetallic CompoundsORCID 0000-0003-3023-1072Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Biochemistry· 1 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Organism:human· 2 Research Focus:metabolism & bioenergetics· 2 Assay:metabolomics· 2 Approach:statistical model· 2 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1

Frequent Co-Authors

Alice ChiodiiD· 2 shared Beatrice CampanellaiD· 2 shared Elena LevantiniiD· 2 shared Emilia BramantiiD· 2 shared Ettore MoscaiD· 2 shared Eva Cabrera San MillániD· 2 shared Giorgia MaroniiD· 2 shared Giovanni Cercignani· 2 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersBiochemistry

Alice ChiodiORCID

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2 papers

Recent·Most cited

BiochemistrybioRxivPreprintJul 4, 2025

Deciphering the Role of Acetate in Metabolic Adaptation and Drug Resistance in Non-Small Cell Lung Cancer

Giorgia Maroni, Eva Cabrera San Millán, Beatrice Campanella…·Emilia Bramanti

This study identifies a metabolic adaptation in osimertinib-resistant NSCLC cells involving acetate production via a PAA shunt, which supports redox balance and bioenergetics.

2 citations

humanbulk RNA-seq

𝕏 PostBluesky

Cancer BiologybioRxivPreprintJun 27, 2025

Reprogramming of Osimertinib-Resistant EGFR-mutant NSCLC: The Pyruvate-Acetaldehyde-Acetate Pathway As a Key Driver of Resistance

Giorgia Maroni, Eva Cabrera San Millán, Raffaella Mercatelli…·Elena Levantini

This study identifies metabolic reprogramming in osimertinib-resistant EGFR-mutant NSCLC, highlighting the pyruvate-acetaldehyde-acetate pathway and mitochondromic alterations as key drivers of resistance.

3 citations

humanbulk RNA-seq

𝕏 PostBluesky