Narendra K. Bairwa

The paper identifies a genetic interaction between SAF1 and RLM1 in Saccharomyces cerevisiae that influences stress response, showing differential tolerance to various stressors.

The paper describes an affordable assay for detecting apoptosis in S. cerevisiae using DAPI staining and Image J to quantify multi-nuclei features and nucleus-to-cytoplasm ratio after treatment with acetic acid and hydrogen peroxide.

The paper investigates the genetic interaction between ATG1 and UCC1 in S. cerevisiae, showing synthetic growth defects and sensitivity to genotoxic agents.

This study shows that deleting YDR131C and ATG1 in yeast causes synthetic growth defects and flocculation, indicating their roles in parallel pathways affecting stress response and growth.

The paper shows that deletion of CTF4 and SAF1 in S. cerevisiae leads to smaller cell size and increased stress tolerance, but sensitivity to HU.

This study shows that the combined absence of SAF1 and CTF8 in S. cerevisiae leads to MMS resistance, HU sensitivity, and genome instability.

The paper investigates the genetic interaction between SAF1 and RRM3 in S. cerevisiae, showing that their combined inactivation causes synthetic growth defects and sensitivity to genotoxic stress.