Author

Young Doo Kim

Baylor College of MedicineORCID 0000-0003-3231-7371Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Organism:mouse· 3 Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Approach:statistical model· 2 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Assay:spatial transcriptomics· 1

Frequent Co-Authors

Huda Yahya ZoghbiiD· 3 shared Bhupesh VaidyaiD· 2 shared Aaran VijayakumaraniD· 1 shared Ajay Kumar VermaiD· 1 shared Annabeth LoftmaniD· 1 shared Benjamin D. W. BelfortiD· 1 shared Benjamin Russell ArenkieliD· 1 shared Cameron OstermaniD· 1 shared

Activity by Year

2026-2026 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 26 mutual co-author links

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3 shared papersNeuroscience

Huda Yahya ZoghbiORCID

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3 papers

Recent·Most cited

NeuroscienceProceedings of the National Academy of SciencesPeer-reviewedAug 10, 2026

Selective loss of Primary Cilia and Neurotrophic Signaling in G51D alpha-Synuclein Mice Highlights a Common Pathway to Parkinsons Disease

Yu‐En Lin, Ebsy Jaimon, Young Doo Kim…·Suzanne R. Pfeffer

The paper shows that G51D alpha-synuclein mice exhibit loss of primary cilia and neurotrophic signaling in specific brain regions, linking ciliary dysfunction to Parkinson's disease pathology.

1 citation

mouse

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NeurosciencebioRxivPreprintJun 8, 2026

Astrocytic ACSBG1 depletion improves lipid-cytokine signaling and attenuates α-Synuclein pathology in a Parkinson's disease mouse model

Young Doo Kim, Bhupesh Vaidya, Jiyoen Kim…·Huda Yahya Zoghbi

This study shows that reducing ACSBG1 in astrocytes reduces inflammation and α-Synuclein pathology in a Parkinson's disease mouse model.

DOI 10.64898/2026.05.20.726454

mousebulk RNA-seq

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NeurosciencebioRxivPreprintMay 19, 2026

GLP-1 analogs restore inflammatory, mitochondrial and intercellular signaling networks in the SncaG51D/G51D knock-in mouse model of Parkinson's disease

Bhupesh Vaidya, Yan Li, Young Doo Kim…·Huda Yahya Zoghbi

GLP-1 analogs improve motor and non-motor deficits and reduce gliosis and insoluble α-synuclein in a Parkinson's disease mouse model.

DOI 10.64898/2026.05.18.726024

mousebulk RNA-seq

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