Author

Carolina Duque

Johns Hopkins UniversityORCID 0000-0003-3737-3858Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Immunology· 1 Microbiology· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Approach:statistical model· 3 Assay:bulk RNA-seq· 2 Research Focus:cell-cell communication· 2 Research Focus:differential expression· 2 Assay:proteomics· 2 Research Focus:benchmarking· 1 Research Focus:cell type annotation· 1

Frequent Co-Authors

Monica R. MugnieriD· 3 shared Edith MálagaiD· 2 shared Jill M. C. HakimiD· 2 shared Robert H. GilmaniD· 2 shared Rony Pedro Colanzi· 2 shared Sneider Alexander Gutierrez GuarnizoiD· 2 shared Aashik Bhalodia· 1 shared Angel Duran· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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3 shared papersMolecular Biology

Monica R. MugnierORCID

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3 papers

Recent·Most cited

Molecular BiologybioRxivPreprintJan 13, 2026

Maternal Trypanosoma cruzi infection is associated with significant placental remodeling regardless of vertical transmission

Sneider Alexander Gutierrez Guarnizo, Jaime So, Carolina Duque…·The Chagas Disease Working Group in Bolivia and Peru

This study shows that maternal Trypanosoma cruzi infection causes significant placental remodeling and systemic changes, regardless of whether the infection is transmitted to the fetus, and identifies potential biomarkers for prenatal monitoring.

DOI 10.64898/2026.01.13.699142

humanbulk RNA-seq

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MicrobiologybioRxivPreprintJun 4, 2025

Clinical Trypanosoma cruzi isolates share a common antigen repertoire that is absent from culture adapted strains

Jill M. C. Hakim, Sneider A.G. Guiterrez, Angel Duran…·Working Group on Chagas Disease in Bolivia and Peru

The paper sequenced clinical T. cruzi isolates, finding genomic instability and a unique antigen repertoire absent in lab-adapted strains.

1 citation

humanwhole-genome sequencing

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ImmunologybioRxivPreprintMay 8, 2024

Splenic follicular B cells promote adverse cardiac remodeling after myocardial infarction via MHC II-dependent antigen presentation.

Obialunanma V. Ebenebe, Jana P. Lovell, Carolina Duque…·Luigi Adamo

This study shows splenic follicular B cells worsen heart failure after MI by presenting cardiac antigens via MHC II, promoting adverse cardiac remodeling.

3 citations

mousehuman

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