Author

Tia J. Kowal

Palo Alto Veterans Institute for ResearchORCID 0000-0003-3879-4718Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Cell Biology· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:clustering· 1 Research Focus:database / resource· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1

Frequent Co-Authors

Alessandro Bulfone· 1 shared Anastasia P. NesterovaiD· 1 shared Benjamin Lawson· 1 shared Biao WangiD· 1 shared Brianna G Feld· 1 shared Catherine A. Priest· 1 shared Chien‐Hui LoiD· 1 shared Chun CheniD· 1 shared

Activity by Year

2024-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperNeuroscience

Alessandro Bulfone

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

NeurosciencebioRxivPreprintDec 3, 2024

Human stem cell-derived GABAergic interneuron development reveals early emergence of subtype diversity followed by gradual electrochemical maturation

Marina Bershteyn, Hongjun Zhou, Luis C. Fuentealba…·Cory R. Nicholas

The paper develops human MGE-pINs from pluripotent stem cells and shows their maturation into SST and PVALB subtypes with functional and molecular characteristics.

4 citations

humanscRNA-seq

𝕏 PostBluesky

Cell BiologybioRxivNov 1, 2024

Defective Neuronal Differentiation in Lowe Syndrome is Associated with Mitochondrial Dysfunction and Impaired Cilia-related Sonic Hedgehog Signaling

Grzegorz Walkiewicz, Siyu Chen, Chien‐Hui Lo…·Yang Sun

This study identifies mitochondrial dysfunction and impaired Sonic Hedgehog signaling as key defects in Lowe syndrome, linking OCRL loss to altered neuronal differentiation and ciliary homeostasis.

DOI 10.7554/elife.104055

humanmouse

𝕏 PostBluesky