Qingnian Goh

This study identifies JNK signaling as a key mediator of neonatal muscle growth and contracture formation, showing its role in myonuclear displacement and muscle size regulation.

This study investigates the role of sympathetic innervation in neonatal muscle growth and contractures, finding it is not required or sufficient for contracture formation but influences muscle width in a sex-specific manner.

This study shows that MSTN signaling inhibition prevents NBPI-induced contractures in female mice but not males, revealing sex-specific effects on muscle growth and proteostasis.

The paper identifies myomerger as a critical protein required for myoblast fusion and demonstrates its role in inducing fusogenicity in non-fusogenic cells.