Author

Thomas M Morin

Massachusetts General HospitalORCID 0000-0003-4120-0669Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Neuroscience· 2

Signature Assays, Organisms & Methods

Click to filter · or open tag

Organism:human· 2 Assay:imaging· 2 Approach:statistical model· 2 Assay:fluorescence / live-cell microscopy· 1 Research Focus:host-pathogen & immune response· 1 Research Focus:synaptic & neural circuits· 1

Frequent Co-Authors

Anne S. BerryiD· 2 shared Hsiang-Yu CheniD· 2 shared William J. JagustiD· 2 shared Joseph GiorgioiD· 1 shared Michael J BreakspeariD· 1 shared Ming M HsuiD· 1 shared Shaian Aghasoltan· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

7 top collaborators · 17 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersNeuroscience

Anne S. BerryORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

NeurosciencebioRxivPreprintNov 6, 2026

Dopamine synthesis capacity and tau pathology differentially modulate rest-to-task functional network dynamics

Thomas M Morin, Hsiang-Yu Chen, Shaian Aghasoltan…·Anne S Berry

This study examines how dopamine synthesis and tau pathology affect functional brain network dynamics during rest and task performance in young and older adults.

DOI 10.64898/2026.11.04.764247

humanfluorescence / live-cell microscopy

𝕏 PostBluesky

NeurosciencebioRxivPreprintJul 10, 2026

Dopamine Compensates for Amyloid-Induced Default Mode Network Dysfunction to Support Learning

Joseph Giorgio, Thomas Morin, Hsiang-Yu Chen…·William J. Jagust

This study shows that dopamine compensates for amyloid-induced dysfunction in the default mode network to support learning in preclinical Alzheimer's disease.

DOI 10.64898/2026.07.07.736872

humanimaging

𝕏 PostBluesky