Krista Metz

This paper identifies GABAB receptors as mediators of presynaptic inhibition in Ia afferents, showing their reduced function after spinal cord injury.

The paper shows that GABAergic neurons facilitate sensory feedback by preventing spike propagation failure at axon branch points through GABAA receptor activity.

The paper investigates the mechanism of extensor H-reflex suppression by flexor afferent conditioning, finding it results from post-activation depression of Ia-afferents and long-lasting inhibition of motoneurons, not direct presynaptic inhibition.

This study shows that sensory and corticospinal pathways facilitate, rather than inhibit, sensory feedback in humans by activating nodal GABAA receptors on Ia-afferents.