Author

Jun Gu

Indiana University IndianapolisORCID 0009-0001-3010-499XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Genomics· 2 Neuroscience· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Organism:rat· 2 Approach:statistical model· 2 Research Focus:cell type annotation· 1 Research Focus:database / resource· 1

Frequent Co-Authors

Chen Ding· 2 shared Gang Wei· 2 shared Guoliang Lv· 2 shared Hongbo Nie· 2 shared Jinfeng Hu· 2 shared Meng Chen· 2 shared Miao Han· 2 shared Ting NiiD· 2 shared

Activity by Year

2015-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersGenomics

Chen Ding

Mutual co-authors in cluster

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3 papers

Recent·Most cited

NeurosciencebioRxivPreprintOct 13, 2023

CD1d-dependent neuroinflammation impairs tissue repair and functional recovery following a spinal cord injury

Xiangbing Wu, Jianyun Liu, Wei Li…·Randy Richard Brutkiewicz

This study shows CD1d promotes proinflammatory responses after spinal cord injury, impairing tissue repair and functional recovery, and identifies CD1d as a potential therapeutic target.

4 citations

mouseexperimental assay

𝕏 PostBluesky

GenomicsbioRxivPreprintDec 2, 2015

Global lengthening of 3′ untranslated regions of mRNAs by alternative cleavage and polyadenylation in cellular senescence

Miao Han, Guoliang Lv, Hongbo Nie…·Ting Ni

This study shows that alternative polyadenylation (APA) leads to longer 3' UTRs in senescent cells, affecting gene regulation through miRNA and RBP binding sites.

DOI 10.1101/033480

mouserat

𝕏 PostBluesky

GenomicsbioRxivPreprintNov 11, 2015

Progressive lengthening of 3′ untranslated regions of mRNAs by alternative cleavage and polyadenylation in cellular senescence of mouse embryonic fibroblasts

Miao Han, Guoliang Lv, Hongbo Nie…·Ting Ni

The paper investigates APA regulation in cellular senescence, finding genes in senescent cells preferentially use distal polyadenylation sites, associated with longer 3′ UTRs and senescence-related pathways.

DOI 10.1101/031302

mouserat

𝕏 PostBluesky