Author

Xiangbing Wu

Indiana University IndianapolisORCID 0000-0001-7207-148XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 1 Neuroscience· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:drug response / drug discovery· 1 Assay:imaging· 1 Assay:proteomics· 1 Organism:rat· 1

Frequent Co-Authors

Wei WuiD· 2 shared Alaa Ibrahim Quddam· 1 shared Alan Yaacoub· 1 shared Arif HussainiD· 1 shared Asim AliiD· 1 shared Chen CheniD· 1 shared Christopher B. Shields· 1 shared Christopher H. Yu· 1 shared

Activity by Year

2023-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 15 mutual co-author links

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2 shared papersPathology

Wei WuORCID

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3 papers

Recent·Most cited

Cell BiologybioRxivPreprintJul 1, 2026

A Nerve-Dependent NGF Receptor Switch Controls Corneal Epithelial Renewal In Neurotrophic Keratopathy

Arif Hussain, Kiana Tajdaran, Ramkumar Katturajan…·Konstantin Feinberg

The paper shows that denervation shifts NGF receptor signaling in corneal epithelial stem cells from TrkA-AKT to p75NTR-JNK, explaining limited rhNGF efficacy in neurotrophic keratopathy and suggesting receptor-selective modulation as a therapy.

DOI 10.64898/2026.06.30.735622

mouseproteomics

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PathologybioRxivPreprintDec 7, 2025

Early blood-spinal cord barrier stabilization preserves peri-lesional tissue and improves recovery after SCI

Chen Chen, Yan Sun, Xiaolong Du…·Wei Wu

This study identifies early vascular leakage beyond the injury epicenter as a driver of secondary pathology after SCI and shows that stabilizing the blood-spinal cord barrier improves recovery.

DOI 10.64898/2025.12.03.692136

ratimaging

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NeurosciencebioRxivPreprintOct 13, 2023

CD1d-dependent neuroinflammation impairs tissue repair and functional recovery following a spinal cord injury

Xiangbing Wu, Jianyun Liu, Wei Li…·Randy Richard Brutkiewicz

This study shows CD1d promotes proinflammatory responses after spinal cord injury, impairing tissue repair and functional recovery, and identifies CD1d as a potential therapeutic target.

4 citations

mouseexperimental assay

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