Anvitha Sriram

This study shows that keratinocytes contribute to mechanical and cold hypersensitivity after traumatic nerve injury and that pannexin-1 activation is critical for this effect.

This study establishes a heat-induced pain model in Fabry disease and identifies a heat shock protein-TRPA1 regulatory axis as a key mechanism.

The paper shows that keratinocyte Piezo1 contributes to paclitaxel-induced mechanical hypersensitivity by sensitizing keratinocytes to mechanical stimulation.