Author

Mitsuyoshi Takatori

University of the RyukyusORCID 0009-0002-7585-3764Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:ATAC-seq· 1 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Approach:experimental assay· 1 Approach:genetic perturbation / CRISPR· 1 Organism:human· 1

Frequent Co-Authors

Chiaki KatagiriiD· 1 shared Chitoshi TakayamaiD· 1 shared Gakuya TakamatsuiD· 1 shared Hirotaka James OkanoiD· 1 shared Kanako Toyama· 1 shared Kaoru KinjoiD· 1 shared Masayuki MatsushitaiD· 1 shared Naoaki OtsukaiD· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCell Biology

Chiaki KatagiriORCID

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1 paper

Recent·Most cited

IF1 restrains excessive elevation of the mitochondrial membrane potential and safeguards the epithelial state of human iPSCs

Kaoru Kinjo, Gakuya Takamatsu, Kanako Toyama, Chitoshi Takayama, Yuko Akamine, Riko Kuniyoshi, Naoaki Otsuka, Yoko Manome, Hirotaka James Okano, Chiaki Katagiri, Mitsuyoshi Takatori, Masayuki Matsushita

This study shows that IF1 limits mitochondrial membrane potential elevation and prevents epithelial-mesenchymal transition in human iPSCs by inhibiting ATP hydrolysis.

bioRxiv · Sep 3, 2026 · PreprintCell Biology