Author

William J. H. Davis

University of AucklandORCID 0009-0003-0870-5419Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Organism:human· 1 Approach:statistical model· 1

Frequent Co-Authors

Aniruddha ChatterjeeiD· 1 shared Catherine J. DrummondiD· 1 shared Cushla McKinneyiD· 1 shared Euan James RodgeriD· 1 shared Glen ReidiD· 1 shared Grégory Gimenez· 1 shared M Thompson· 1 shared Mark E. HatleyiD· 1 shared

Activity by Year

2026-2026 · peak 1/yr

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10 top collaborators · 45 mutual co-author links

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1 shared paperCancer Biology

Aniruddha ChatterjeeORCID

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1 paper

Recent·Most cited

Cancer BiologybioRxivPreprintMay 12, 2026

Elevated Expression of MALAT1 Contributes to the Survival of Drug-Tolerant Persister Cells Following Targeted Therapy in Lung Adenocarcinoma

William J. H. Davis, M Thompson, Safia M Farry…·Glen Reid

This study identifies MALAT1 as a key lncRNA supporting drug-tolerant persister cells in lung adenocarcinoma, showing its role in treatment resistance and suggesting it as a potential therapeutic target.

DOI 10.64898/2026.05.07.723110

humanbulk RNA-seq

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