Mark E. Hatley

This study uses a PAX3::FOXO1 inducible model to show P3F initiates ARMS transformation but is not required for maintaining proliferation or transcriptional programs.

This study shows that RNA quality control through rny mutations enables antibiotic tolerance in Streptococcus pneumoniae by balancing transcriptional resilience and degradation.

This study identifies MALAT1 as a key lncRNA supporting drug-tolerant persister cells in lung adenocarcinoma, showing its role in treatment resistance and suggesting it as a potential therapeutic target.