Author

Alexis M. Hayes

Florida International UniversityORCID 0009-0004-9591-6050Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Bioinformatics· 3

Signature Assays, Organisms & Methods

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Research Focus:differential expression· 3 Organism:human· 3 Approach:statistical model· 3 Approach:experimental assay· 2 Research Focus:gene regulatory network inference· 2 Assay:DNA methylation· 1 Assay:proteomics· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

Amber Delmas EliasoniD· 3 shared David P. MarcianoiD· 3 shared Santiago Moreno-CaceresiD· 3 shared

Activity by Year

2026-2026 · peak 3/yr

Co-Author Collaboration Network

3 top collaborators · 3 mutual co-author links

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3 shared papersBioinformatics

Amber Delmas EliasonORCID

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3 papers

Recent·Most cited

COPD Preferentially Disrupts Constitutive Redox Support Relative to Inducible Antioxidant Defense

Alexis M. Hayes, Santiago Moreno-Caceres, Amber Delmas Eliason, David P. Marciano

The paper identifies a cell-selective loss of constitutive redox support in COPD, with reduced PGC-1 target-program output in fibroblasts.

bioRxiv · Oct 8, 2026 · PreprintBioinformatics

8-Oxoguanine transcriptional miscoding can transiently reproduce PAH-associated premature-stop transcripts

Santiago Moreno-Caceres, Alexis M. Hayes, Amber Delmas Eliason, David P. Marciano

The paper shows that 8-oxoguanine can create premature-stop transcripts in PAH genes, mimicking pathogenic variants without DNA mutation.

bioRxiv · Oct 8, 2026 · PreprintBioinformatics

Disease-associated protein states accessible to 8-oxoguanine transcriptional miscoding without genomic mutation

Santiago Moreno-Caceres, Alexis M. Hayes, Amber Delmas Eliason, David P. Marciano

The paper identifies disease-associated protein states caused by 8-oxoG transcriptional miscoding, showing 5.7% of pathogenic missense variants can arise without genomic mutation.

bioRxiv · Oct 4, 2026 · PreprintBioinformatics

Alexis M. Hayes · henhao