Deborah D’Aliberti

This study shows that Pcdh9 deletion in mice causes synaptic overgrowth and excitatory transmission defects in the hippocampus, revealing its role in synaptic morphology and function.

The paper identifies hemogenic endothelium in vitelline and umbilical arteries as a major source of fetal lympho-myeloid progenitors in mice, showing their transient contribution before definitive HSCs emerge.

The paper shows that SET levels influence enhancer usage during cell fate acquisition, leading to altered gene regulation and contributing to disease.

The paper analyzes SARS-CoV-2 synonymous mutations across 400,000 samples, showing that mutations increasing codon similarity to human codons tend to persist, suggesting adaptation to human host codon usage.