Alana Maerivoet

This study shows medin induces pro-inflammatory and prothrombotic changes in endothelial cells and is strongly associated with coronary plaque burden and inflammation.

This paper evaluates AlphaFold 3 and other methods in predicting α-synuclein polymorphs using a reference set of known structures and finds they can generate fibril-like models, though performance varies with template use.

This study shows that Medin induces pro-inflammatory activation in human brain vascular smooth muscle cells via NF-kappaB, without affecting structural proteins involved in phenotypic transformation.