Neha E. H. Dinesh

This paper investigates how fibronectin mutations impair chondrogenesis in skeletal dysplasia by disrupting protein secretion and causing cellular stress in mesenchymal stem cells.

This study investigates the role of fibronectin isoforms in postnatal skeletal development using knockout mice, finding that combined deletion of both isoforms leads to skeletal defects and altered chondrogenesis.