Author

Koki Saito

Hirosaki UniversityORCID 0009-0007-5348-7739Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 1 Microbiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1 Organism:fungi (other)· 1

Frequent Co-Authors

Eiji MoritaiD· 1 shared Eishin IwaoiD· 1 shared Hirotoshi SushidaiD· 1 shared Kazuya MaedaiD· 1 shared Mai Ohara· 1 shared Mariko KoudaiD· 1 shared Masashi ArakawaiD· 1 shared Takumi KawaseiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperCell Biology

Eiji MoritaORCID

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2 papers

Recent·Most cited

Cell BiologybioRxivPreprintOct 9, 2026

Secretion is a second fate for p62 and is coupled to KEAP1/NRF2 signaling

Koki Saito, Tomohiro Takahashi, Masashi Arakawa, Eiji Morita

This study shows p62 can exit cells through secretion, a process linked to KEAP1/NRF2 signaling and distinct from autophagy.

DOI 10.64898/2026.10.07.757498

humanproteomics

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MicrobiologybioRxivPreprintNov 24, 2025

Transcriptomic insights into the tritrophic plant-pathogen-mycoparasite interaction reveal coordinated reprogramming fungal secretomes and plant amino acid metabolism.

Kazuya Maeda, Mariko Kouda, Mai Ohara…·Yuichiro Iida

This study examines the tritrophic interaction between tomato, the pathogen C. fulvum, and the mycoparasite H. pulvinata, revealing coordinated changes in gene expression that suppress the pathogen and induce plant resistance.

DOI 10.1101/2025.11.24.690098

plant (other)fungi (other)

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