Author

A. B. Wass

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Microbiology· 3 Physiology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 4 Research Focus:host-pathogen & immune response· 3 Organism:human· 3 Research Focus:cell signaling & trafficking· 2 Research Focus:gene regulatory network inference· 2 Assay:bulk RNA-seq· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1

Frequent Co-Authors

C. M. O'Connor· 3 shared B. A. Krishna· 2 shared A. I. Salameh· 1 shared B. W. Geiler· 1 shared D. E. Huffmann· 1 shared F. J. Moss· 1 shared G. C. Chan· 1 shared G. Gros· 1 shared

Activity by Year

2020-2023 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 17 mutual co-author links

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3 shared papersMicrobiology

C. M. O'Connor

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Virion-associated US28 rapidly modulates Akt activity to suppress HCMV lytic replication in monocytes

J. Mahmud, B. W. Geiler, J. Biswas, M. J. Miller, J. E. Myers, S. M. Matthews, A. B. Wass, C. M. O'Connor, G. C. Chan

This study shows virion-associated US28 modulates Akt activity to suppress HCMV lytic replication in monocytes, enabling quiescent infection.

bioRxiv · Sep 5, 2023 · PreprintMicrobiology

Role of channels in the oxygen permeability of red blood cells

P. Zhao, R. R. Geyer, A. I. Salameh, A. B. Wass, S. Taki, D. E. Huffmann, H. J. Meyerson, G. Gros, R. Occhipinti, F. J. Moss, W. F. Boron

The paper shows that oxygen crosses red blood cell membranes via protein channels, not just lipid diffusion, using experiments and genetic knockouts to identify key channels involved.

bioRxiv · Aug 28, 2020 · PreprintPhysiology

Activator protein-1 transactivation of the major immediate early locus is a determinant of cytomegalovirus reactivation from latency

B. A. Krishna, A. B. Wass, C. M. O'Connor

This study identifies AP-1 transactivation of the MIE enhancer as critical for HCMV reactivation from latency, showing its role in de-repressing viral transcripts and downstream genes.

Proceedings of the National Academy of Sciences · Aug 11, 2020Peer-reviewedMicrobiology

The Requirement For US28 During Cytomegalovirus Latency Is Independent Of US27 And US29 Gene Expression

B. A. Krishna, A. B. Wass, R. Sridharan, C. M. O'Connor

This study shows US28 is required for HCMV latency, independent of US27 and US29 gene expression, using mutant viruses in hematopoietic cells.

bioRxiv · Jan 19, 2020Microbiology