Author

A. C. Bianco

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 3

Signature Assays, Organisms & Methods

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Organism:mouse· 3 Assay:bulk RNA-seq· 2 Research Focus:chromatin & epigenetic regulation· 2 Approach:statistical model· 2 Assay:ATAC-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Assay:DNA methylation· 1 Approach:experimental assay· 1

Frequent Co-Authors

G. W. Fernandes· 2 shared A. Collins· 1 shared B. Gereben· 1 shared B. M. L. C. Bocco· 1 shared C. Gu· 1 shared C. Wei· 1 shared F. Salas-Lucia· 1 shared I. C. Kettelhut· 1 shared

Activity by Year

2020-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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2 shared papersPhysiology

G. W. Fernandes

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Author profile →

3 papers

Recent·Most cited

Truncated suPAR simultaneously causes kidney disease and autoimmune diabetes mellitus

K. Zhu, K. Mukherjee, C. Wei, S. S. Hayek, A. Collins, C. Gu, K. Corapi, M. M. Altintas, Y. Wang, W. S. Sushrut, A. C. Bianco, J. Reiser, S. Sever

The paper identifies a truncated suPAR fragment, D2D3, that causes kidney disease and autoimmune diabetes by impairing pancreatic beta-cell function and kidney health.

bioRxiv · Apr 26, 2022 · PreprintPhysiology

NEONATAL THYROXINE ACTIVATION MODIFIES EPIGENETIC PROGRAMMING OF THE LIVER

T. L. Fonseca, T. Garcia, G. W. Fernandes, T. M. Nair, A. C. Bianco

This study shows that neonatal thyroxine activation permanently alters liver epigenetic programming by modifying DNA methylation and chromatin accessibility in adult mice.

bioRxiv · Dec 7, 2020Physiology

The transcriptional repressor Zfp125 modifies hepatic energy metabolism in response to fasting and insulin resistance

G. W. Fernandes, B. M. L. C. Bocco, T. d. L. Fonseca, F. Salas-Lucia, O. Nickel, S. C. Russo, B. Gereben, I. C. Kettelhut, A. C. Bianco

This study shows that Zfp125 regulates hepatic energy metabolism by modulating gene expression related to fatty acid oxidation, ketogenesis, and gluconeogenesis, with implications for type 2 diabetes.

bioRxiv · Jul 2, 2020 · PreprintPhysiology