Author

A. E. Davies

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 1 Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1

Frequent Co-Authors

A. R. Ram· 1 shared C. Juliano· 1 shared D. M. Zuckerman· 1 shared E. M. Langer· 1 shared G. Quon· 1 shared H. R. Helms· 1 shared I. C. McLean· 1 shared J. Copperman· 1 shared

Activity by Year

2018-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 13 mutual co-author links

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1 shared paperCancer Biology

A. R. Ram

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Author profile →

3 papers

Recent·Most cited

A Single-Cell Bioprinting Method to Reconstruct Native Cellular Microenvironments with Subcellular Resolution

H. R. Helms, K. A. Oyama, A. E. Davies, E. M. Langer, L. E. Bertassoni

This paper introduces a single-cell bioprinting method that reconstructs native cellular microenvironments with subcellular resolution, enabling precise interrogation of spatial interactions.

bioRxiv · Feb 1, 2024 · PreprintBioengineering

Single-cell morphodynamical trajectories enable prediction of gene expression accompanying cell state change

J. Copperman, I. C. McLean, S. M. Gross, J. Singh, V. Murthy, Y. H. Chang, A. E. Davies, D. M. Zuckerman, L. M. Heiser

The paper develops MMIST, a computational approach linking live-cell morphodynamics to gene expression changes during cell state transitions.

bioRxiv · Jan 18, 2024Cell Biology

Systems-level properties of EGFR/Ras/ERK signalling amplify local signals to generate gene expressionplasticity

A. E. Davies, T. E. Gillies, S. Siebert, M. Pargett, S. J. Tobin, A. R. Ram, C. Juliano, G. Quon, M. J. Bissell, J. G. Albeck

The paper shows that EGFR/Ras/ERK signaling in invasive breast cancer cells produces more dynamic and disordered ERK activity, leading to heterogeneous gene expression compared to non-malignant cells.

bioRxiv · Nov 9, 2018 · PreprintCancer Biology