Author

M. J. Bissell

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 1 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Assay:proteomics· 2 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

A. E. Davies· 1 shared A. J. Kent· 1 shared A. R. Ram· 1 shared C. Hochman-Mendez· 1 shared C. Juliano· 1 shared C. Robertson· 1 shared G. Quon· 1 shared J. G. Albeck· 1 shared

Activity by Year

2018-2019 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCancer Biology

A. E. Davies

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2 papers

Recent·Most cited

The microstructure of laminin-111 compensates for dystroglycan loss in functional differentiation of mammary epithelial cells

A. J. Kent, N. Mayer, J. L. Inman, C. Hochman-Mendez, M. J. Bissell, C. Robertson

The paper investigates whether polymerized laminin-111 can compensate for dystroglycan loss in mammary epithelial cell differentiation by forming functional networks.

bioRxiv · Jan 25, 2019Bioengineering

Systems-level properties of EGFR/Ras/ERK signalling amplify local signals to generate gene expressionplasticity

A. E. Davies, T. E. Gillies, S. Siebert, M. Pargett, S. J. Tobin, A. R. Ram, C. Juliano, G. Quon, M. J. Bissell, J. G. Albeck

The paper shows that EGFR/Ras/ERK signaling in invasive breast cancer cells produces more dynamic and disordered ERK activity, leading to heterogeneous gene expression compared to non-malignant cells.

bioRxiv · Nov 9, 2018 · PreprintCancer Biology