Author

A. Fidanza

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 3

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Approach:experimental assay· 3 Research Focus:cell signaling & trafficking· 2 Organism:human· 2 Research Focus:cell-cell communication· 1 Assay:CRISPR screen· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

A. May· 2 shared F. P. Luongo· 2 shared H. A. Taylor· 2 shared N. Romano· 2 shared T. Ventura· 2 shared H. McClafferty· 1 shared H. Volmer· 1 shared J. J. Bieker· 1 shared

Activity by Year

2019-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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2 shared papersCell Biology

A. May

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Author profile →

3 papers

Recent·Most cited

A novel human pluripotent stem cell-based gene activation system identifies IGFBP2 as a mediator in the production of hematopoietic progenitors in vitro

P. Petazzi, T. Ventura, F. P. Luongo, H. McClafferty, A. May, H. A. Taylor, M. Shipston, N. Romano, L. Forrester, P. Menendez, A. Fidanza

This paper introduces an iCRISPRa system to activate transcription factors in iPSCs, identifying IGFBP2 as critical for hematopoietic progenitor production.

bioRxiv · Jan 14, 2024 · PreprintCell Biology

Modelling the Erythroblastic Island Niche of Dyserythropoietic Anaemia Type IV patients using Induced Pluripotent Stem Cells

A. May, T. Ventura, A. Fidanza, H. Volmer, H. A. Taylor, N. Romano, S. L. D'Souza, J. J. Bieker, L. M. Forrester

This study uses iPSCs to model CDA type IV, showing KLF1-E325K mutations impair erythroid cell production and slightly affect macrophage maturation in the EBI niche.

bioRxiv · Feb 2, 2023Cell Biology

Late Embryogenesis Abundant (LEA) proteins confer water stress tolerance to mammalian somatic cells

M. Czernik, A. Fidanza, F. P. Luongo, L. Valbonetti, P. A. Scapolo, P. Patrizio, P. Loi

This study demonstrates that LEA proteins protect mammalian somatic cells from desiccation injury by maintaining cell viability and mitochondrial function during dehydration and rehydration.

bioRxiv · Jul 16, 2019Cell Biology